[Advanced-Only] Seroquel

[motorheadlk]

There was a guy in a /mlp/ thread that said that he took seroquel to slow down a meth high, the thing is that he took it without knowing it was an anti-psichotic, which made his tulpa disappear. 4 days later she appeared again, no trauma, no memory loss, no form issues and no personality deviations. She said that she was in a dream-like state.

 

This made me wonder, if this drug temporaly erases the "weaker" consciousness in a brain, what would happen if a tulpa that is switched took it, would the host disappear for some time? What with a tulpa that is already imposed and is really strong, would she disappear too?

 

What I mean to say is, does this drug make the weaker consciousness disappear, or does it make the consciousness that is not of the host disappear? If a person that has switched with his tulpa take it, would he regain control and his tulpa would disappear or would he disappear for some time?

 

That's only speculation and the risks are too high for anyone to try (unless you're mad for science). I just created this thread to debate the subject and see if anyone here is crazy enough to consider doing that (which probably won't happen since so little people achieved switching).

 

Not only for the debate, I'm posting this as a reminder that there is a test that could help people into knowing if a tulpa that is able to switch is in equal place to the hosts consciousness or if she's still in a way, for not being the native consciousness, "weaker" than the hosts consciousness. That could point to a tulpa being equal or weaker than the host, so I'm curious to what would happen. What do you guys think that would happen?

[glitchthe3rd]

Well, antipsychotics are designed to suppress hallucinations, after all. And guess what tulpae are?

[motorheadlk]

Well, antipsychotics are designed to suppress hallucinations, after all. And guess what tulpae are?

 

So, in your opinion, if a tulpa that has switched makes the body ingest the drug, she would disappear and the host would take control back?

Or were you just being sarcastic? D:

 

[glitchthe3rd]

I don't know that taking an antipsychotic would suppress a switched tulpa. In all likelihood the host would be suppressed.

[penetratenkos]

Anti-Psychotics block D2 receptors, according to Wikipedia. I don't really know if this could challenge the theory that a Tulpa is located in the subconscious, but what I do know is that this is up for debate. I wouldn't really consider anything fact until we can get a Neurologist to the scene.

[Mabus]

I would postulate that atypical antipsychotics have an effect on tulpas, specifically one mediated at 5HT2a. Why 5HT2a? Atypical antipsychotics are 5HT2a antagonists, and there appears to be some anecdotal evidence that tulpas are affected negatively by them. I notice that Anika seems more real when I forget to take my mirtazapine and aripiprazole (both 5HT2a antagonists). It is important to note that I am taking those medications for clinical depression, not for a schizoid disorder, because there is no psychosis (which might make my tulpa seem more real) to suppress. Additionally, both myself and others have noted significant success with the 5HT2a agonists psilocybin and psilacetin.

 

Essentially, it appears as though 5HT2a agonists positively impact tulpas, 5HT2a antagonists negatively impact tulpas, and both have the potential to cause tulpas to disappear temporarily. Of course, without more evidence, this all just amounts to speculation and theory. It is also important to note that both atypical antipsychotics and psilocybin have an effect on dopaminergic systems, and that correlation≠causation.

 

Edit:

I wouldn't really consider anything fact until we can get a Neurologist to the scene.

 

Actually, I think that a pharmacologist might be more helpful with respect to antipsychotics.

[penetratenkos]

and there appears to be some anecdotal evidence that tulpas are affected negatively by them.

The nature this evidence may not qualify as scientific evidence because it cannot be trustfully tested. It also cannot be tested using the scientific method. This information is used by the word of mouth, and although evidence is presented in a scientific matter, anecdotal evidence cannot be used as validating evidence.

[Mabus]

The nature this evidence may not qualify as scientific evidence because it cannot be trustfully tested. It also cannot be tested using the scientific method. This information is used by the word of mouth, and although evidence is presented in a scientific matter, anecdotal evidence cannot be used as validating evidence.

 

Which is precisely why I explicitly labeled it as postulation and then stuck a disclaimer on the end. Additionally, I can personally vouch for the validity of multiple reports, as 2 of them come directly from me, and the events reported in another were supervised by me.

[penetratenkos]

 

Which is precisely why I explicitly labeled it as postulation and then stuck a disclaimer on the end.

I thought the disclaimer was tongue-in-cheek, like the disclaimer on South Park. Thank you for the clarity.

[motorheadlk]

I would postulate that atypical antipsychotics have an effect on tulpas, specifically one mediated at 5HT2a. Why 5HT2a? Atypical antipsychotics are 5HT2a antagonists, and there appears to be some anecdotal evidence that tulpas are affected negatively by them. I notice that Anika seems more real when I forget to take my mirtazapine and aripiprazole (both 5HT2a antagonists). It is important to note that I am taking those medications for clinical depression, not for a schizoid disorder, because there is no psychosis (which might make my tulpa seem more real) to suppress. Additionally, both myself and others have noted significant success with the 5HT2a agonists psilocybin and psilacetin.

 

Essentially, it appears as though 5HT2a agonists positively impact tulpas, 5HT2a antagonists negatively impact tulpas, and both have the potential to cause tulpas to disappear temporarily. Of course, without more evidence, this all just amounts to speculation and theory. It is also important to note that both atypical antipsychotics and psilocybin have an effect on dopaminergic systems, and that correlation≠causation.

 

Edit:

 

Actually, I think that a pharmacologist might be more helpful with respect to antipsychotics.

 

Of course, it makes sense on why a tulpa would disappear after the host ingest a antipsychotic, still the doubt about what would happen in the case that they switched (Who would disappear) remains unclear.

---

The nature this evidence may not qualify as scientific evidence because it cannot be trustfully tested. It also cannot be tested using the scientific method. This information is used by the word of mouth, and although evidence is presented in a scientific matter, anecdotal evidence cannot be used as validating evidence.

 

Every evidence about everything we didn't try for ourselves in the tulpa phenomenon is anedoctal, as we still couldn't figure out how to scientifically prove the existence of a self-imposed consciousness on a healthy brain (see the thread "Proof of tulpae").

 

Despite this, we are able to gather intel and do research about it ourselves if we want to, and that's only possible by sharing experiences and techniques of the phenomenon. There's no need to state that we don't have scientific evidence for something by saying that there are only reports of people saying what happens to them, since the whole tulpa belief system is based on the sharing of experiences between the hosts.

 

Trying to explain what we found through our empiric experiences is, however, a need, to try and understand the whole phenomenon, and speculate on what is possible and what is not, if the tulpa business was taken as something supernatural and the like, many people that are here wouldn't be, I know I'm one of them.

 

The objective of this thread is, above anything else, improve our current model and clear beliefs that are contradicting, specifically the ones that claim that tulpae are or aren't conscious in the same way we are, just different by the same level my consciousness and yours would be.

---

Anti-Psychotics block D2 receptors, according to Wikipedia. I don't really know if this could challenge the theory that a Tulpa is located in the subconscious, but what I do know is that this is up for debate. I wouldn't really consider anything fact until we can get a Neurologist to the scene.

 

I don't think this would challenge the theory, as even I believe that our own consciousness rose from the subconscious.

 

The only thing I think this test could help with is to understand if the differences we observe between the tulpa consciousness and the hosts consciousness is purely a matter of position (the term waffles used on our last debate about this) or there is something else to it. I'm one that believe that our consciousness is not the same to that of a tulpa, what differs us, however, is not something I can simply describe, since we barely know our own consciousness and how it differs from someone else's consciousness.